Ph128
You have given me a daunting task of condensing volumes of medical information into a brief and, hopefully, helpful response. I recognize your obvious intelligence and your genuine concern and, for a lack of a better word, 'grief', over your current health. I believe a broader understanding of hypertension and the treatment and sequelae will be helpful for you in understanding why I have answered as I did--that more likely than not, your care was rendered at or above the minimal acceptable standard of care (that is all that is required).
I'll start with your last two questions,
Would I have still suffered damage if my condition were adequately treated? It wasn’t, why not?
Would you have? Who knows. Could you have? Yes; and, more than likely not, would have.
I can not assume (and neither should you or your cardiologist) that your hypertension was not managed properly (at or above the minimum standard of care). A cardiologist, obviously educated and skilled at a higher degree in cardiovascular systems than is a family physician, and especially a cardiologist much younger than your former family physician, can not be used as the measuring stick for evaluating the performance of your former family physician. The cardiologist may truly believe that if he had been managing your hypertension this would not have happened; but in reality, a cardiologist isn't going to provide routine management of essesntial (primary) hypertension and if he did, the cardiologist could only manage the BP and could not manage the unknown factor(s) producing hypertension.
Hypertension is the result of increased cardiac output (overworking of the heart muscle, which leads to enlargement of the cardiac muscle and to reduced ejection fractions) and increased peripheral vascular resistance. The mechanisms that control cardiac output and peripheral vascular resistance are the Central Nervous System, the renal (kidney) pressor system (renin-angiotensin-aldosterone system), and extracellular fluid volume (fluid outside of the cells, aka third-spacing, aka edema or swelling). Why these mechanisms fail is not known. The medications you received correct, or attempt to correct, the failed action(s) by the CNS, renal pressor system, and extracellular fluid volume.
Did I have ECGs in the past? Yes, from my previous physician whom I was with for about 9 years. He had me take one about every 1 ½ years. He said my condition was difficult to treat at his office due to White Coat Syndrome. He said he couldn’t get good bp readings and the ECGs would allow him to monitor if his treatment was adequate. He said my bp was labile. He prescribed beta blocker and ACE for control. My current family doctor continued with the same meds but later change them to CCB and ACE. I tried to find him (for my records) but his office is closed now. He is probably retired.
Apparently, your ECG showed no problem during these nine years.
It is important to know that receiving an ECG with or without additional medical intervention, more likely than not, would not have prevented cardiac muscle enlargement and decreased ejection fraction.
Through medication, your heart muscle was given protection against overwork and enlargement. There is little difference in the medications prescribed by the two family physicians and your cardiologist. Beta-blockers reduce cardiac output by competitively blocking the number of impulses from the sympathetic nervous sytem to the heart muscle (blocking the beta-adrenergic receptors). Calcium-channel blockers inhibit the movement of calcium ions across the membranes of the cardiac and arterial muscle cells, which results in the depression of impulse formation in specialized cardiac pacemaker cells, which leads to decreased cardiac work (and decreased cardiac energy consumption, and increased delivery of oxygen to the heart's cells). ACE (angiotensin-converting enzyme) inhibitors work in the lungs to stop ACE from converting to angiotensin I, which is activated when renin is released from the kidneys. Simply put, ACE inhibitors reduce extracellular fluid levels.
So, each family physician gave you medication to prevent cardiac enlargement, which results in decreased ejection fractions, and gave you medication to reduce extracellular fluid levels.
The cardiologist sent me through about 15 thousand dollars worth of tests (including Hospital for heart cath). I suspect he was trying to rule in or out just what was causing my problem. My bp was not good. He didn’t buy the White Coat Syndrome after seeing my condition. His diagnoses, after all the tests, poorly controlled hypertension. He does have it regulated now with 4 different medications. (coreg, ACE, CCB, ARB).
"Poorly controlled hypertension" is a medical term. In medical-speak, "poorly controlled" means even with (proper) medication, diet, exercise, the hypertension remained labile (unstable, ie. poorly controlled). It is not a statement of physician performance evaluation.
Coreg is an alpha- and beta-blocking agent to reduce cardiac output. The beta-blocking part assists in preventing reflux tachycardia assosciated with alpha-blockers and decreases plasma renin activity (extracellular fluid management). ARB (angiotensin II receptor blockers) work in the vascular smooth muscle and in the adrenal gland (both controlled by the CNS) to block the binding of angiotensin II. (Remember ACE inhibitors prevent the formation of angiotensin II from angiotensin I). So, these new meds work differently yet the same as the mediations you previously received.
Third paragraph: No one has ever told me my LVH could be caused by the meds I was taking. Perhaps I was not taking them. What meds would do that? And then again you say an ECG would give valuable information to show if such were happening.
Don't worry; from the meds you listed, you did not receive any of the medications to which I referred.
Fourth: Showing signs of LVH? I was not showing major signs of LVH. It must have come on gradually. It was found when I had another condition (paracarditis) and was x-rayed. That’s when I was referred to the cardiologist. My family doctor was very surprised when viewing the x-ray.
Pericarditis is not a minor player in this picture! Either the origin of the pericarditis, the pericarditis, or both, exacerbated the weakening of your heart muscle and decreasing ejection fraction.
Did I suffer damage that I wouldn’t have suffered anyway? Is it above the minimum standards to wait until a major crisis to take place to then realize a course of treatment was failing? Perhaps an ECG would not have been the tool of choice to see if the treatment was working. Which tool? That is probably what my question should be. Why wasn’t it used?
It would be very difficult to reasonably prove that you suffered significant damage that you would not have otherwise suffered. Each year's standards of practice would have to reviewed and compared against the care you received in that year. Then, it would have to be determined when you knew or should have known that something was wrong. As your first indication that something was wrong occurred during a hospitalization for pericarditis, it will be even harder to prove that you had signs and symptoms of additional problems that were overlooked, undiagnosed, and untreated, which led to your developing cardiac enlargement and decreased/inadequate ejection fractions. (I'm determined to type 'injection fractions', so if I missed correcting any, please forgive me.)
Perhaps a different diagnostic test such as a cardiac cath would have given you a chance to receive additional treatment; but apparently, your physical findings did not indicate the need for such. Were you short of breath? easily fatigued? unable to enjoy your usual activities due to shortness of breath, chest pain, heart palpitations? Were your feet and legs (ankles) swelled and, or edematous? Could you push your thumb into any edema and leave a depressed (1/2-2"") impression of your thumb in the edema? If the answers are "no", then you most likely did not have clinical findings indicative of cardiac output deficit and therefore no indication for a cardiac catheterization.
There are many physical conditions that can not be and are not diagnosed until such time as the condition produces a crisis. I wish it were not so. Hopefully one day in the near future the general public will have available to them the 'body scan' that a select few have received (as seen and discussed on 'Oprah" a couple of years ago). That technology is truly preventative medicine. It detects subtle changes that would not have produced symptoms for decades.
I hope I have answered your questions satisfactorily. I urge you to meet with an medical malpractice attorney in your area. The consultation visits are usually free and certainly, the appointment will be beneficial to you. A statute of limitations applies to medmal claims and you must not let the statute expire or you are forever barred from bringing a medmal action against the physician. You also have the issue of the physician-in-question's being retired. I truly believe a consultation appointment will satisfactorily answer many of your questions.
Best wishes,
EC